GAITHERSBURG, Md., Nov. 2, 2016 /PRNewswire/ -- MaxCyte, Inc., a developer and supplier of cell engineering products and services to biopharmaceutical firms engaged in cell therapy, drug discovery and development, biomanufacturing, gene editing and immuno-oncology, announced that Debra K. Bowes has joined the Company as Executive Vice President, Business and Strategic Development. Ms. Bowes has more than 25 years' experience in corporate strategy, licensing and in the creation of partnerships to advance the development and commercialization of biopharmaceutical products, with a main emphasis in oncology. Before coming to MaxCyte, Ms. Bowes was interim President and Chief Executive Officer of CapGenesis Pharma, in Bethesda, MD. Previously, she served as President and Founder of Chevy Chase BioPartners, LLC, a strategic planning consultancy, as well as in leadership positions at CBLI Pharmaceuticals, MedImmune, Amylin Pharmaceuticals, Pfizer, Ligand Pharmaceuticals, Centocor and Hybritech. She also has served as national president of Women in Bio. Ms. Bowes is a Master's Degree candidate at Johns Hopkins University, and has a B.S. in cell biology from the University of Cincinnati. "We're excited to have Deb Bowes join our team at what is an important inflection point for MaxCyte and our CARMA-based programs," said MaxCyte President & CEO Doug Doerfler. "We are advancing our collaborative research programs with CARMA, and are seeking new alliances in this space. Deb will be an invaluable member of our team focused, in particular, on relationships with partners to further develop potential new immuno-oncology drug candidates." In her new role at MaxCyte, Ms. Bowes will lead the strategic planning and execution of all business development activities related to MaxCyte's CARMA-based programs. CARMA, MaxCyte's proprietary cell engineering platform technology, allows simple and rapid manufacture of advanced cancer treatments that utilize a patient's own immune system and is differentiated from traditional chimeric antigen receptor ("CAR") therapy due to its use of mRNA to engineer immune cells delivered back into a patient.