LUND, Sweden, February 1, 2013 /PRNewswire/ -- NeuroVive, a leading mitochondrial medicine company, is pleased to announce the appointment of Jan Nilsson as Chief Operating Officer (COO). Jan will oversee the day-to-day operations of the Company and support the planned commercialisation of CicloMulsion® and NeuroSTAT®, NeuroVive's leading products for the treatment of acute cardiovascular and neurological conditions through mitochondrial protection. Jan has over 30 years of experience in senior management, commercial and sales roles in the pharmaceutical and biotechnology sectors in Sweden and internationally. Since 2011 Jan has served as Managing Director of Lipopeptide AB and as Chief Commercial Officer at Pergamum AB, two clinical-stage Swedish biotech companies developing innovative wound treatments based on therapeutic peptides. Between 2004 and 2009 Jan served as Managing Director of Tripep AB, a listed Swedish-biotech company developing therapeutic DNA-vaccines against hepatitis B and C. Prior to that Jan spent several years at Schering-Plough where he served in a number of senior management, marketing and sales positions, including Vice-President for the Nordic region between 2000 and 2004. Jan has served on the Board of Directors of NeuroVive since 2011. As a result of his new role, Jan will not be standing for re-election to the NeuroVive Board at the next Annual Meeting. CicloMulsion® and NeuroSTAT® are in clinical development by NeuroVive for the treatment of heart reperfusion injury and traumatic brain injury respectively. These are indications of huge medical need for which there are currently no approved pharmaceutical treatment options. Both products act to prevent the death of mitochondria in damaged cells and the cascade of intracellular biochemical events that lead to secondary tissue damage following a traumatic injury. By protecting a cell's mitochondria NeuroVive's products ensure that energy production is preserved and a damaged cell's normal regenerative mechanisms can act to repair and maintain the cell.